I am going to do an experiment. I want to decipher if I have a yeast issue or if it is histamines that are making me itch. So, the idea is that if I take anti-histamine drug it will quell the histamines, but it wouldn't touch the itching if it is due to yeast. Would this be true? So, what is the specific (real scientific) that yeast does to cause the itching--does the yeast cause a release of histamine? If you have yeast overload but not allergies, would histamine stop the itching of the yeast?

-Rachel

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I'm not sure. But, I learned that scratching the surface of the skin causes the body to release histamines to that location. When folks do 'dry brushing' for ASD kids, or for detox protocols, I wonder how that 'helps'.

If you have a wound/scratch/abrasion, that is not an allergenic response. But, the tightness of the skin as it heals or the breaking of the skin edges?? often causes itching also. My understanding is that the histamine reaction is part of the body's healing process.

It seems that there are various reasons that histamines are released at the surface of the skin. The yeast, could be itching merely from friction and abrasion against clothing, or from other body parts rubbing together - such as an armpit or the fold behind the knee.

If you scratch, it will itch more as the histamines are released, from what I have learned and experienced.

Not sure on the various epithelial functions of the epidermis and all of that stuff anymore. A dermatologist may be able to provide a more scientific explanation of the anatomy and physiology process.

Found this, not sure exactly what it means, lol. It appears that there is some thinning of the epidermis with some histamine blocker medication (h1 blockers and h2 blockers). But, that histamine isn't the variable causing the thickening??? Maybe??
http://www3.interscience.wiley.com/journal/119555092/abstract?CRETR...

My best guess of the practical relevance of this is not related to yeast directly, however. With asthma, they use different h1 and h2 blocker medication to make the airways less inflammatory. However, asthmatics also seem to become more sensitive to inhaled irritants. A cascade of issues develop for asthmatics who become dependent on the h1 and h2 blockers. Basically, we remove the body's healing response (send histamine to the surface of the brochials), but without removing the stimulant, the body develops a more and more reactive airway.

Benedryl is an h1 blocker
Tagamet the antacid is an h2 blocker.
There are h1 and h2 blocker tricyclic antidepressants.
H2-receptors cause an increase in heart rate and contractility.
(The histamine response is important to many body functions. I wonder what the repeated and daily use of antihistamines does to alter the normal functions of the body, especially, its ability to heal itself!)

Basically, histamine blockers "anti-histamines" just suppress the reactions of the body, not address the underlying cause of the inflammatory process. I took antihistamines daily from about age 12-36.

Funny that changing my diet around age 36, addressed the symptoms (daily headaches, sinus issues, excess mucus in the throat) and the antihistamines were no longer required. Ever - for 10 years now.

Yeast is a balancing response of the body to an insufficiency in the beneficial gut microbials. When we have adequate nutrients the 'good guys' survive in the gut, and yeast becomes obsolete, unnecessary. When we don't have adequate nutrients, the toxins build up, the 'good guys' die off and are replaced with the yeast trying to eat up the excess anti-nutrients such as excess sugar, bind mercury, release toxins faster (ie. through the leaky gut). The toxins leak through the gut and must excrete through the skin (an 'overflow system' to rid the body of excessive toxins). Ultimately, it appears that the fungal growth could become either localized as in a cancer, or systemic as an autoimmune disorder, from my understanding.


Pat

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