Forum Discussions (502)

Discussions Replies Latest Activity

VDR Taq (vitamin D)

This is a vitamin D receptor. There is a mutation that makes it harder to get vitamin D. VDR Taq, COMT and BH4 work together to influence…

Started by Shannon

0 Feb 13, 2009


COMT degrades dopamine and norepinephrine. It can be slowed down with mutation V158M and H62H, or if you are low on methyl groups. Low COM…

Started by Shannon

0 Feb 13, 2009

MTRR (B12)

MTRR recharges B12 by adding a methyl group to it. This enzyme can be downregulated. blood B12 is not a good marker. blood homocysteine ma…

Started by Shannon

0 Feb 13, 2009

MTR (B12 & meat)

MTR uses methyl B12 to convert folate and homocysteine into methionine. It can be upregulated (A2756G) which results in a deficiency of met…

Started by Shannon

0 Feb 13, 2009

SHMT (folate)

This enzyme is needed to convert folic acid (as opposed to folate) into the precursor that MTHFR uses. Mutation C1420T makes it work slowe…

Started by Shannon

0 Feb 13, 2009


This enzyme pulls homocysteine from the methyl cycle to initiate the transsulfuration cycle. It works faster in some people than in others.…

Started by Shannon

0 Feb 13, 2009

Enzymes & Methylation

Steps through each of the enzymes and explains what they do and how to overcome. Yeah, complicated, but good explanations: http://heartfixe…

Started by Shannon

0 Feb 13, 2009

Krebs cycle (citric acid cycle)

Involves glucose, glycolysis, acetyl-coenzyme A and makes ATP. toxins and Krebs cycle (and homeopathy):

Started by Shannon

1 Feb 13, 2009
Reply by Nessa

Detoxification in the liver

Started by Shannon

1 Feb 12, 2009
Reply by kortner

methylation nutrient cascade

This goes in reverse. Find your deficiency, and everything above it is blocked. Methyl groups are donated (by SAM) to bind to toxins. SAM…

Started by Shannon

0 Feb 12, 2009



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